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Attention-deficit/hyperactive disorder updates.
Kessi M, Duan H, Xiong J, Chen B, He F, Yang L, Ma Y, Bamgbade OA, Peng J, Yin F. Kessi M, et al. Front Mol Neurosci. 2022 Sep 21;15:925049. doi: 10.3389/fnmol.2022.925049. eCollection 2022. Front Mol Neurosci. 2022. PMID: 36211978 Free PMC article. Review.
RESULTS: Several genes have been reported to associate with ADHD: DRD1, DRD2, DRD4, DAT1, TPH2, HTR1A, HTR1B, SLC6A4, HTR2A, DBH, NET1, ADRA2A, ADRA2C, CHRNA4, CHRNA7, GAD1, GRM1, GRM5, GRM7, GRM8, TARBP1, ADGRL3, FGF1, MAOA, BDNF, SNAP25, STX1A, ATXN7, and SORCS2. …
RESULTS: Several genes have been reported to associate with ADHD: DRD1, DRD2, DRD4, DAT1, TPH2, HTR1A, HTR1B, SLC6A4, HTR2A, DBH, NET …
Molecular genetics of attention-deficit/hyperactivity disorder: an overview.
Banaschewski T, Becker K, Scherag S, Franke B, Coghill D. Banaschewski T, et al. Eur Child Adolesc Psychiatry. 2010 Mar;19(3):237-57. doi: 10.1007/s00787-010-0090-z. Epub 2010 Feb 10. Eur Child Adolesc Psychiatry. 2010. PMID: 20145962 Free PMC article. Review.
In recent years a large number of studies on different candidate genes for ADHD have been published, most have focused on genes involved in the dopaminergic neurotransmission system, such as DRD4, DRD5, DAT1/SLC6A3, DBH, DDC. Genes associated with the noradrenergic (such a …
In recent years a large number of studies on different candidate genes for ADHD have been published, most have focused on genes involved in …
Molecular heterosis: a review.
Comings DE, MacMurray JP. Comings DE, et al. Mol Genet Metab. 2000 Sep-Oct;71(1-2):19-31. doi: 10.1006/mgme.2000.3015. Mol Genet Metab. 2000. PMID: 11001792 Review.
A number of examples are reviewed, including those for the following genes: ADRA2C, C3 complement, DRD1, DRD2, DRD3, DRD4, ESR1, HP, HBB, HLA-DR DQ, HTR2A, properdin B, SLC6A4, PNMT, and secretor. ...
A number of examples are reviewed, including those for the following genes: ADRA2C, C3 complement, DRD1, DRD2, DRD3, DRD4, ESR …
Clinical and molecular genetics of ADHD and Tourette syndrome. Two related polygenic disorders.
Comings DE. Comings DE. Ann N Y Acad Sci. 2001 Jun;931:50-83. doi: 10.1111/j.1749-6632.2001.tb05773.x. Ann N Y Acad Sci. 2001. PMID: 11462757 Review.
Some of the specific loci involved are dopamine genes--DRD2, DRD4, DRD5, and the dopamine transporter; norepinephrine (NE) and epinephrine (EPI) genes--dopamine beta-hydroxylase, ADRA2A, ADRA2C, PNMT, norepinephrine transporter, MAOA, COMT; serotonin genes--TDO2, HT …
Some of the specific loci involved are dopamine genes--DRD2, DRD4, DRD5, and the dopamine transporter; norepinephrine (NE) and epinep …
An in-frame deletion in the alpha(2C) adrenergic receptor is common in African--Americans.
Feng J, Zheng J, Gelernter J, Kranzler H, Cook E, Goldman D, Jones IR, Craddock N, Heston LL, Delisi L, Peltonen L, Bennett WP, Sommer SS. Feng J, et al. Mol Psychiatry. 2001 Mar;6(2):168-72. doi: 10.1038/sj.mp.4000817. Mol Psychiatry. 2001. PMID: 11317218
In addition, ADRA2C binds clonidine which is prescribed for three psychiatric diseases. ...TIDRU(1) occurs at a location similar to the third intracytoplasmic 48-nucleotide repeat unit in the DRD4 that is associated with ADHD. Although these data do not suggest an a …
In addition, ADRA2C binds clonidine which is prescribed for three psychiatric diseases. ...TIDRU(1) occurs at a location similar to t …
GPCR Pharmacological Profiling of Aaptamine from the Philippine Sponge Stylissa sp. Extends Its Therapeutic Potential for Noncommunicable Diseases.
Luyao H, Luesch H, Uy M. Luyao H, et al. Molecules. 2021 Sep 16;26(18):5618. doi: 10.3390/molecules26185618. Molecules. 2021. PMID: 34577088 Free PMC article.
Dose-response analysis validated the alpha-adrenoreceptor antagonist activity of aaptamine (ADRA2C, IC(50) 11.9 M) and revealed the even more potent antagonism of the beta-adrenoreceptor (ADRB2, IC(50) 0.20 M) and dopamine receptor D4 (DRD4, IC(50) 6.9 M). ...
Dose-response analysis validated the alpha-adrenoreceptor antagonist activity of aaptamine (ADRA2C, IC(50) 11.9 M) and revealed the e …
Altered Expression of Genes Encoding Neurotransmitter Receptors in GnRH Neurons of Proestrous Mice.
Vastagh C, Rodolosse A, Solymosi N, Liposits Z. Vastagh C, et al. Front Cell Neurosci. 2016 Oct 7;10:230. doi: 10.3389/fncel.2016.00230. eCollection 2016. Front Cell Neurosci. 2016. PMID: 27774052 Free PMC article.
Differential gene expression was most apparent in GABA-ergic (Gabbr1, Gabra3, Gabrb3, Gabrb2, Gabrg2), glutamatergic (Gria1, Gria2, Grin1, Grin3a, Grm1, Slc17a6), cholinergic (Chrnb2, Chrm4) and dopaminergic (Drd3, Drd4), adrenergic (Adra1b, Adra2a, Adra2c), adenosi …
Differential gene expression was most apparent in GABA-ergic (Gabbr1, Gabra3, Gabrb3, Gabrb2, Gabrg2), glutamatergic (Gria1, Gria2, Grin1, G …
The additive effect of neurotransmitter genes in pathological gambling.
Comings DE, Gade-Andavolu R, Gonzalez N, Wu S, Muhleman D, Chen C, Koh P, Farwell K, Blake H, Dietz G, MacMurray JP, Lesieur HR, Rugle LJ, Rosenthal RJ. Comings DE, et al. Clin Genet. 2001 Aug;60(2):107-16. doi: 10.1034/j.1399-0004.2001.600204.x. Clin Genet. 2001. PMID: 11553044
Fifteen genes were included in the regression equation. The most significant were the DRD2, DRD4, DAT1, TPH, ADRA2C, NMDA1, and PS1 genes. The r(2) or fraction of the variance was less than 0.02 for most genes. ...
Fifteen genes were included in the regression equation. The most significant were the DRD2, DRD4, DAT1, TPH, ADRA2C, NMDA1, an …